Background/aims: Abnormal levels of electromyographic activity (EMG) are often observed in muscles affected by stroke, likely reflecting altered motor unit recruitment and discharge patterns (1, 2). However, post-stroke clinical assessments, such as Fugl-Meyer (FM) motor scales and Berg Balance Scale (BBS), may not sensitively detect neuromuscular alterations (3).This study examined the influence of EMG on outcomes assessed by commonly used post-stroke clinical scales.
Methods: Sixteen individuals with chronic stroke (59.0±13.9 years; 28.3±23.1 months post-stroke) were assessed using upper-extremity (UEFM), lower-extremity (LEFM) and total FM (TFM), as well as BBS scores. Surface EMG (root mean square over 250 ms) was recorded from the paretic deltoid and rectus femoris muscles during shoulder abduction and flexion, and hip flexion and knee extension maximal voluntary isometric contractions using a Neuro-EMG-Micro-4 system. Relationships between clinical and EMG variables were examined by correlation and regression analyses.
Results: TFM correlated with shoulder abduction (r=0.76, p<0.001), shoulder flexion (r=0.66, p=0.006), hip flexion (r=0.51, p=0.04), and knee extension EMG (r=0.82, p<0.001). LEFM correlated with knee extension EMG (r=0.74; p<0.001), whereas UEFM correlated with shoulder abduction (r=0.78, p<0.001) and shoulder flexion EMG (r=0.64, p=0.008). BBS correlated with knee extension EMG only (r=0.56, p=0.03).
Conclusion: Shoulder abduction and flexion EMG explained 52% of UEFM, knee extension EMG explained 45% of LEFM and 14% of BBS, and all EMG measures explained 81% of TFM variances. As clinical scales do not directly assess impaired neural drive, EMG may serve as an objective biomarker for quantifying neuromuscular dysfunction and monitoring impairment after stroke.